All documents are PDF. If needed, you can download Adobe reader here. 
CNBP regulates forebrain formation at organogenesis stage in chick embryos
Haploinsuffciency for Znf9 in Znf9+/− Mice Is Associated with Multiorgan Abnormalities Resembling Myotonic Dystrophy
The Triple Functions of D2 Silencing in Treatment of Periapical Disease
C1 Silencing Attenuates Inflammation and Alveolar Bone Resorption in Endodontic Disease
Molecular cloning, developmental expression, promoter analysis and functional characterization of the mouse CNBP gene
Bone resorption deficiency affects tooth root development in RANKL mutant mice due to attenuated IGF-1 signaling in radicular odontoblasts
The zinc-finger protein CNBP is required for forebrain formation in the mouse
Inhibition of Rgs10 Expression Prevents Immune Cell Infiltration in Bacteria-induced Inflammatory Lesions and Osteoclast-mediated Bone Destruction
TRAF Family Member-Associated NF-κB Activator (TANK) Induced by RANKL Negatively Regulates Osteoclasts Survival and Function
Silencing of Atp6v1c1 Prevents Breast Cancer Growth and Bone Metastasis
Chondrocyte-specific Knockout of Cbfβ Reveals the Indispensable Function of Cbfβ in Chondrocyte Maturation, Growth Plate Development and Trabecular Bone Formation in Mice
Ga13 negatively controls osteoclastogenesis through inhibition of the Akt-GSK3b-NFATc1 signalling pathway (See graphic at the bottom of the page)
Core binding factor beta (Cbfβ) controls the balance of chondrocyte proliferation and differentiation by up-regulating Indian hedgehog (Ihh) expression and inhibiting parathyroid hormone-related protein Receptor (PPR) expression in postnatal cartilage and bone formation
C/ebpα controls osteoclast terminal differentiation, activation, function, and postnatal bone homeostasis through direct regulation of Nfatc1
Monocyte-specific knockout of C/ebpα results in osteopetrosis phenotype, blocks bone loss in ovariectomized mice and reveals an important function of C/ebpα in osteoclast differentiation and function.
V-ATPase subunit ATP6AP1 (Ac45) regulates osteoclast differentiation, extracellular acidification, lysosomal trafficking, and protease exocytosis in osteoclast-mediated bone resorption
Specificity of RGS10A as a key component in the RANKL signaling mechanism for osteoclast differentiation
Deletion of Core-binding factor β (Cbfβ) in mesenchymal progenitor cells provides new insights into Cbfβ/Runxs complex function in cartilage and bone development
Ac45 silencing mediated by AAV-sh-Ac45-RNAi prevents both bone loss and inflammation caused by periodontitis
Cbfβ deletion in mice recapitulates cleidocranial dysplasia and reveals multiple functions of Cbfβ required for skeletal development
RNAi-Mediated Silencing of Atp6i and Atp6i Haploinsufficiency Prevents Both Bone Loss and Inflammation in a Mouse Model of Periodontal Disease
CCAAT/Enhancer-binding Protein (C/EBP) Is Important for Osteoclast Differentiation and Activity
Requirement of the Epithelium-specific Ets Transcription Factor Spdef for Mucous Gland Cell Function in the Gastric Antrum
RNA Interference-Mediated Silencing of Atp6i Prevents Both Periapical Bone Erosion and Inflammation in the Mouse Model of Endodontic Disease
Osteoclasts
We study osteoclast gene differentiation, function, positive and negative regulation, and signaling pathways.
