Xiaoying Wang, MD, PhD

Professor, Department of Neurosurgery and Neurology

Program Director, Brain Injury Research, Clinical Neuroscience Research Center
Phone
504-988-2646
Office Address
JBJ Building, Room 613
School or College
School of Medicine

Education & Affiliations

Postdoctoral Research Fellow, Massachusetts General Hospital/Harvard Medical School
PhD, Biophysics, Peking University Health Science Center, China
MD, Henan Medical University

Biography

Dr. Xiaoying Wang is currently Professor of Neurosurgery and Neurology, Program Director of Brain Injury Research at the Clinical Neuroscience Research Center (CNRC) of Tulane University School of Medicine. He joined Massachusetts General Hospital/Harvard Medical School in 1998 as a Postdoctoral Research Fellow (under the supervision of Professor Eng H. Lo) and later was promoted as Associate Director of the Neuroprotection Research Laboratory in 2006, and Associate Professor of Harvard Medical School in 2012. Dr. Wang’s research focuses on experimental investigation of molecular pathophysiology following cerebrovascular diseases and traumatic brain injury (TBI), and translational therapeutic strategy development. Dr. Wang is an established Principal Investigator, had led several NIH RO1 and one translational NIH UO1 grant. His research is currently supported by two NIH RO1s. Dr. Wang has published over 140 research articles in professional journals. Additionally, Dr. Wang serves on a number of national and international research grant review panels such as NIH and American Heart Association. He also serves as an editorial board member for numerous professional journals, including Brain Research, Neurological Research, Stroke, PLOS ONE, Translational Stroke Research, and Frontiers Neurology.

Publications

Complete List of Published Work in MyBibliography:

https://www.ncbi.nlm.nih.gov/myncbi/xiaoying.wang.1/bibliography/public/

 

 

Dr. Wang’s main research interests are in understanding the role of aging in ischemic stroke, particularly in molecular pathophysiology of cerebrovascular dysfunction, neurovascular uncoupling, silent cerebrovascular disease, and vascular dementia.